Chem Biol Interact 2003, 143–144:551–558.CrossRefPubMed 16. Wang HY, Yan MY, Zhao YW, Kan B: Transcriptional repressor gene – mtlR of mannitol PTS operon in Vibrio

cholerae. Wei Sheng Wu Xue Bao 2007, 47:522–525.PubMed 17. Yebra MJ, Veyrat A, Santos MA, Martinez GP: Genetics of L-Sorbose Transport and Metabolism in Lactobacillus casei. J Bacteriol 2000, 182:155–163.CrossRefPubMed 18. Watanabe S, Hamano M, Kakeshita H, Bunai K, Tojo S, Yamaguchi H, Fujita Y, Wong SL, IWP-2 ic50 Yamane K: Mannitol-1-phosphate dehydrogenase (MtlD) is required for mannitol and glucitol assimilation in Bacillus subtilis : possible cooperation of mtl and gut operons. J Bacteriol 2003, 185:4816–4824.CrossRefPubMed 19. Takahashi N, Abbe K, Takahashi-Abbe S, Yamada T: Oxygen sensitivity of sugar metabolism and interconversion of pyruvate formate-lyase in intact cells SAR302503 of Streptococcus mutans and Streptococcus sanguis. Infect Immun 1987, 55:652–656.PubMed 20. Olivier V, Haines GK, Tan Y, Satchell KJ: Hemolysin and the multifunctional autoprocessing

RTX toxin are virulence factors during intestinal infection of mice with STA-9090 supplier Vibrio cholerae El Tor O1 strains. Infect Immun 2007, 75:5035–5042.CrossRefPubMed 21. Olivier V, Salzman NH, Satchell KJ: Prolonged colonization of mice by Vibrio cholerae El Tor O1 depends on accessory toxins. Infect Immun 2007, 75:5043–5045.CrossRefPubMed 22. Williams SG, Varcoe LT, Attridge SR, Manning PA:Vibrio cholerae Hcp, a secreted protein coregulated

with HlyA. Infect Immun 1996, 64:283–289.PubMed 23. Williams SG, Attridge SR, Manning PA: The transcriptional activator HlyU of Vibrio cholerae : nucleotide sequence and role in virulence gene expression. Mol Microbiol 1993, 9:751–760.CrossRefPubMed Authors’ contributions RW carried out the main part of experiments in this study and drafted the manuscript, HZ carried out qRT-PCR and participated in discussion in preparing the manuscript, HQ participated in cultures and sample preparation, SG and BK designed and coordinated the study, and BK revised the manuscript. All authors read and approved the final manuscript.”
“Background Extraintestinal pathogenic E. coli (ExPEC) strains are implicated in a large number of infections in humans and animals, such click here as urinary tract infection (UTI), meningitis, diverse intraabdominal infection, pneumonia, osteomyelitis, and soft-tissue infection; besides, bacteremia can accompany infection at any of these sites. ExPEC, which include avian pathogenic (APEC) E. coli, uropathogenic E. coli (UPEC), septicemic E. coli, and newborn meningitis-causing E. coli (NMEC), exhibit considerable genome diversity characterized by the possession of various combinations of adhesins (e.g., P and S fimbriae), iron-acquisition systems (e.g., aerobactin), host defense-avoidance mechanisms (e.g., capsule or O-specific antigen), toxins (e.g.